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Amniotic Fluid Increased by Homoeopathy



Hpathy Ezine, August, 2012 | Print This Post Print This Post |

A woman having her first child shows a low amniotic fluid index. After homeopathic treatment, normal levels were found to be restored.

Mrs S.L

Age – 18 Yrs

Sex – Female

Date – 04/12/2011

History –

She came with a history of 33 weeks ANC primigravida.

LMP – 04/02/2010

Her EDD was – 07/01/12.

USG – Obstetrics ( 03/12/2011)

Amniotic Fluid Index (AFI) 5.3, ( Normal is 8-18 )

The rest was normal

O/E – Fetal movement normal

No edema over feet / face.

Generalities:-

Appetite – Normal

Desires – Salty, Sour, Apples

Aversion – Sweet, Milk

Perspiration – Face

Sleep – Normal

Dreams – Unremembered

Past History – Typhoid

Thermals – Hot

Mental Generals –

Cowardice

Anger – indisposed to talk

Nervous Temperament

Cannot express herself.

Remedy –Lycopodium 200 2 doses stat.

Advised to take protein rich diet and plenty of water.

Follow up:

09/12/2011:- REVIEW USG OBSTETRICS (08/12/2011)

AFI – 10.8

Remedy – S.L

28/12/2011:- USG Obstetrics (19/12/2011)

AFI – 11.6

Remedy – Carcinocin 200 2 doses. (Anti – miasmatic).

05/01/2012: Full term ANC.

Fetal Movements Normal

Lumbar pain since 2 days.

Remedy –Caulophyllum 30 TDS

Follow up with Obstetrician for further assessment and management.

Patient underwent FTND on 15/01/2012 and delivered a healthy female baby.

USG KUB of Baby: ( 16/01/2012 )

Impression – Normal Study

Scanned copies of all USG Reports are attached below serially

USG After 5 days of treatment

Documents below:

USG After 15 days of treatment

Comments: - This was a case of 33 weeks ANC and all other modern faculty doctors were insisting on the patient having elective induction of labor and SOS LSCS delivery as her AFI was 5.3. However homoeopathy had the ability to increase AFI as early as possible. 2 doses of Lycopodium were given at intervals of one hour.

Follow up intercurrent was anti miasmatic: Carcinocin 200 2 doses. It was given for removal of any miasmatic blocks in the baby.

After delivery USG of Baby was done to confirm for any congenital Anomaly in the baby.

Oligohydramnios and Homoeopathy

Definition:-Oligohydramnios is a condition in which there is not enough amniotic fluid surrounding and cushioning the baby. It usually develops in the latter part of the third trimester.

Incidence:-About 8 percent of pregnant women will experience low amniotic fluid levels, although only 4 percent will be diagnosed with Oligohydramnios. Women are at increased risk for the condition if they go past their due date as amniotic fluid levels decrease by nearly 50 percent after week 42. It causes complications in 12 percent of pregnant women past week 41

Amniotic fluid:-Amniotic fluid is to be found in the amniotic cavity. It completely surrounds the embryo after the 4th week of pregnancy. The amniotic fluid is a clear, watery fluid that is filtered out of the maternal blood via the amniotic epithelium into the amniotic cavity. The makeup of the amniotic fluid is thus quite complex, with many maternal and fetal constituents. The main constituents are water and electrolytes (99%) together with glucose, lipids from the fetal lungs, proteins with bactericide properties and flaked-off fetal epithelium cells.

Amniotic fluid physiology:-Adequate amniotic fluid volume is maintained by a balance of fetal fluid production (lung liquid and urine) and resorption (swallowing and intramembranous flow). Even though different hypotheses have been advanced on the mechanisms regulating this turnover, the inflow and outflow mechanism that keeps amniotic fluid volume within the normal range is not entirely clear. Regulatory mechanisms act at three levels: placental control of water and solute transfer; regulation of inflows and outflows from the fetus; and, maternal effect on fetal fluid balance. Amniotic fluid is 98-99% water.

About 4 liters of water accumulate within intrauterine compartments during the 40-week period of human gestation, with 2800 ml in the fetus, 400 ml in the placenta, and 800 ml in the amniotic fluid. At the beginning of pregnancy, amniotic fluid volume (AFV) is a multiple of fetal volume. The two volumes become equal soon after the 20th week, but by the 30th week AVF is about half the fetal volume and at term it is about a quarter of fetal volume. In the last trimester, near term, there are net increases of about 30 to 40 ml per day.

Regulatory mechanisms act at three levels:

– Placental control of water and solute transfer.

– Regulation of inflows and outflows from the fetus: fetal urine flow and composition are modulated by arginine, vasopressin, aldosterone, angiotensin II, and atrial natriuretic peptide, in much the same way as they in adults. The mechanisms regulating fetal swallowing are less known.

– Maternal effect on fetal fluid balance: there is a strong relationship between maternal plasma volume expansion during pregnancy and AVF, so that subnormal plasma volume expansion is associated with Oligohydramnios and elevated plasma volume with hydramnios. Kilpatrick et al. (11) reported that ingestion of 2 liters of water in women with a low amniotic fluid index (AFI) resulted in a significant 31% AFI increase.

Causes of Oligohydramnios:-

Ruptures:-A slight tear or rupture in the amniotic sac can cause a gush of fluid or a slow trickle. Premature rupture of the membranes (PROM), where the sac breaks after week 37 but before labor starts, can also result in low amniotic fluid levels.

Mothers Health:-If the mother develops health complications during pregnancy, such as gestational diabetes, pregnancy induced hypertension, Intra-uterine infection and preeclampsia; these can all be possible factors. Any pre-pregnancy conditions, such as diabetes can also be a cause. This is why, when preparing for pregnancy, any existing health concerns are addressed and properly managed.

Sanjay Padole

Dr. Sanjay Gavaji Padole M.D "“ graduated from Dr. Babasaheb Ambedkar Marathwada University and Shri Bhagwan Homoeopathic medical college, Aurangabad. He worked as Resident medical officer at Seth Nandlal Dhoot Hospital Aurangabad and trained at Homoeopathic Education And Research Institute, Dr Kishor Mehta. Vileparle, Mumbai. Since 2003 he"™s has had his own clinic at Mit hospital Cidco, n-4, Aurangabad,Maharashtra, India. His special interests include Indian mythology.

Comments

  1. dr sacheen khirid

    August 17, 2012

    a superb article, very well written & most importantly i like the way Dr S Padole has given clear guidelines of how long to wait & what all to watch out in cases like this.
    cases like this with scientific guidelines will go a long way in helping the neophytes especially answering their queries on the scope of Homeopathy
    Congrats to Dr Padole & team Hpathy for a good write up

  2. Profile photo of butterblu

    butterblu

    August 17, 2012

    This is an unusual case in affecting a specific criteria….but doing it holistically. We could never explain such results to an allopath.

  3. Dr.Naba Kishore Mahapatra

    August 20, 2012

    Thank you Dr.Padole for this AFI information with homeo treatment.What can be done if one has both diabetes and hypertension before pregnancy?

  4. Dr. meenakshi Ambwani

    August 21, 2012

    It is a remarkable work done by you doctor. This will go a long way in helping other homeopaths by expanding their knowledge in these cases .

  5. ganesan.m

    November 2, 2012

    Sir,
    I had met this low level of Amniotic Fluid in my second daughter’s delivery some 15 years back. We went hospital for check up as there was no sign of delivery pain at all. Dr.said that no pain will commence since low level of amniotic fluid condition.I brought my daughter to home and searched through repertory and materia medica and as specific, I found under NAT_M. I remember that Igave three doses of nat_m 30 one afternoon and pain began at about 7 p.m. and my daughter was admitted and she gave birth male child with normal delivery.
    ganesan.m

    • Profile photo of Firuzi Mehta

      Firuzi Mehta

      November 4, 2012

      Dear Mr. Ganesan,
      can you please show us the exact rubrics you found or which Materia Medica you found it in? Thanks!

      Firuzi Mehta

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