The lungs are continually exposed to a number of injurious substances, such as irritant gases discharged into the atmosphere and many kinds of airborne organic and inorganic particles. Severe pulmonary injury may lead to pulmonary fibrosis. Fibrous thickening of alveolar septa makes the lungs increasingly rigid, restricting normal respiratory excursions.
Diffusion of oxygen and carbon dioxide between alveolar air and pulmonary capillaries also is hampered because of the increased thickness of the alveolar septa. Pulmonary fibrosis causes progressive disability similar to that encouraged in pulmonary emphysema.
Some types of collagen diseases, characterized by injury to connective tissue, may have as their major manifestation injury to the connective tissue framework of the lung, leading to pulmonary fibrosis.
Certain occupational diseases are recognized as being caused by inhalation of injurious substances. The general term pneumoconiosis is used to refer to lung injury produced by inhalation of injurious dust or other particulate material. The best known of the pneumoconiosis are silicosis and asbestosis. Silicosis is a type of progressive nodular pulmonary fibrosis caused by inhalation of rock dust. Asbestosis is a diffuse pulmonary fibrosis caused by inhalation of asbestos fibers.
Inhalation of coal dust, cotton fibers, certain types of fungus spores, and many other substances attending certain occupations also may cause pulmonary fibrosis.
Pulmonary fibrosis symptoms and differential diagnosis
Early pulmonary fibrosis is often asymptomatic. Symptoms of pulmonary fibrosis include dyspnea (initially on exertion, then at rest in later stages.) cough, usually nonproductive, chest discomfort/tightness, and fatigue. Clinical signs include crackles on auscultation of the chest, usually at the lung bases and signs of pulmonary hypertension (if associated).
Diagnosis of pulmonary fibrosis
Laboratory tests – routine blood tests are normal. Blood gases may show hypoxemia. Weakly positive titers of antinuclear antibodies and rheumatoid factor are present in a quarter.
Pulmonary function tests show reduction in all lung volumes with impaired gas transfer.
Chest X-ray – may show diffuse predominantly basal and peripheral ground glass change with loss of definition of the heart borders and hemidiaphragms.
Histological examination may show typical cellular infiltrate or fibrosis.
Pulmonary fibrosis treatment with homeopathic medicines
Homeopathy is one of the most popular holistic systems of medicine. The selection of remedy is based upon the theory of individualization and symptoms similarity by using holistic approach. This is the only way through which a state of complete health can be regained by removing all the sign and symptoms from which the patient is suffering. The aim of homeopathy is not only to treat pulmonary fibrosis but to address its underlying cause behind pulmonary fibrosis and individual susceptibility. As far as therapeutic medication is concerned, several remedies are available for the treatment of symptoms of pulmonary fibrosis that can be selected on the basis of cause, sensations and modalities of the complaints. For individualized remedy selection and treatment, the patient should consult a qualified homeopathic doctor in person. There are following remedies which are helpful in the treatment of pulmonary fibrosis symptoms:
Arsenic Album, ipecauc, Natrum Sulph, Phosphorous, Antim Tart, Cuprum Met, Nux Vomica, Stannum Met, Causticum, Sepia, Silicea, Hepar Sulph, Lachesis, Kali Sulph, Kreosote, Arsenic Iod, Kali Bi, Belladonna, Bryonia Alba, Rhus Tox, Tuberculinum, and many other medicines.