Clinical Cases

Hematemesis from Esophageal Varices – Pediatric Emergency

Written by Navin Pawaskar

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Preliminary History: Male child, 8 yrs, middle class

Acute History:

An 8yr old male, only child of educated parents, was brought to the Homeopathic Hospital OPD carried in by his father. This family had never taken homeopathy before. The chief complaint was of frank and large hematemesis. He had had a history of similar episodes in the past, every year about the same time.  Sclerotherapy had not helped prevent these bleeding episodes. They recurred periodically every year.

The acute picture was as below:

Frank profuse bright red bloody vomiting 3- 4  times a day

Passing dark tarry stools 3-4 days prior to vomiting

No history of abdominal pain

No history of having consumed any substance

H/O He had a history of 3 similar episodes in the past. The sequelae, inspite of conventional management was circulatory collapse with dyspnoea and he had to be   revived with blood transfusion and life support systems (ventilators and ICU management).  Each episode lasted for three to four days, before he recovered enough to be taken out of ICU.

Past history

Obstructive jaundice at the age of 1 yr

Milestones delayed. He was a chilly child and craved sweets.

k/c/o Childhood Cirrhosis with varices and episodes of hematemesis


Category II Triage

General condition satisfactory, clammy skin

Heart rate 130/min

Respiratory rate 43/min

Per abdomen : Liver and spleen palpable.


•          TC                                  :         10,000

•          Hb                                  :         4 gm%

•          Platelet                             :         1.4 Lakhs

•          B.T                                 :         4.14/sec.

•          C.T                                 :         3.10/sec.


Gr. II – III Oesophageal varices with portal hypertension due to cirrhosis of liver and splenomegaly presenting with hematemesis and impending hypovolemic shock.


The focus for constructing the totality in this emergency is based on the history obtained of sequelae to these same set of symptoms in the past. There are no characteristics to indicate a simillimum except the annual recurrence of symptoms. The patho-physiology behind the episode that leads to circulatory collapse and shock would hence be important components of the remedy to be chosen. The constitutional symptoms help for further differentiation of a group of remedies covering the pathological presentation. Hence the totality consists of:

1)     Annual aggravation

2)     Location, liver, blood

3)     Pathology and degeneration, destruction, hemorrhage, shock

4)     Craving sweets

5)     Chilly patient

Susceptibility assessment

Pace of disease        :  moderate

Pathology                : degenerative liver- irreversible with complication of varices

Characteristics : few, only pathological symptoms

Sensitivity                : low

Dominant miasm       : tubercular (bleeding, degeneration, shock) with syphilitic background.

Correspondence        : based on pathological generals, venom group

Plan of Emergency Management:

Given this assessment, we see that the vitality is very low and susceptibility is very low. He needs to be revived at any time if he slips into circulatory collapse; he still remains in a compensated phase though he could decompensate anytime. The choice of potency hence is low, single dose and very cautious repetition to avoid fatal aggravation that may present either with increased hematemesis or decompensated circulatory collapse. The remedy should also cover the tubercular /syphilitic miasmatic pathophysiology.

Crot. horridus 30C, cautious repetition

Assessment after each dose

Ancillary Measures:

All precautions to prevent circulatory collapse:

Head low position
Intact IV line

IV infusions to maintain plasma volume

Follow up:

He had only one episode of vomiting after the dose of Crot. H 30C. He had no vomiting the following days. His heart rate reduced and peripheral circulation stabilized thus preventing his further deterioration into hypovolemic shock.  He had no complaints thereafter. He was followed up for more than one year after this to watch for repeat episodes of hematemesis. He had no further episodes, which meant that the annual aggravation had also been prevented.

Discussion DC26:

Establishing Clinico-pathological correlations:

Pathology progression, degeneration of liver (cirrhosis),  destruction of muscular layer of circulatory (venous) system due to backpressure  at varices resulting in hemorrhage and shock.

Knowledge of this should be correlated with the same in terms of remedy pathogenesis. Snake remedies have a similar pathological evolution and hence are indicated in this pathology.

Copperwaithe states: “GENERAL ANALYSIS.

Like other serpent poisons, Crot. acts primarily upon the cerebro-spinal nerve centers, producing secondarily decomposition of the blood, a destruction of blood fibrine, hemorrhages, ecchymosis, etc

Importance of the Time dimension / evolution in the case:

The case itself has presented in a stage of irreversible pathology. Treatment is basically symptomatic and palliative. The focus is to prevent further bleeding from the varices and prevent incipient shock.

Quick clinical assessment and monitoring of vital parameters:

In the emergency presentation the focus is on observing and controlling the onset and development of shock. Appropriate vigilance and monitoring of the heart rate, blood pressure and hematemesis (loss of blood volume) with knowledge of ancillary measures required are important parts of management.


Dr. Navin Pawaskar, MD (Hom)
Director, Clinical services,
ML Dhawle Trust and Organizations

About the author

Navin Pawaskar

Navin Pawaskar
Dr Navin Pawaskar
Director, Ariv Integrative Healthcare & JIMS Healthcare
JIMS Hospital, Hyderabad.
[email protected]
Mobile: +91 750 62 63 508

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