Clinical Cases

Traumatic Suppurative Eye – Threat to Vision

gupta mar image

Dr. Rakesh Gupta treats a patient with a serious eye injury, using both acute and constitutional treatment.

Abstract

This is a unique case which enables us to broaden our scope for treating emergencies related to very vital organs like the eye. It teaches us the importance of understanding disease and the application of homoeopathy. It also demonstrates the concept of pathology, pathogenesis and tissue affection, which enables us to understand the disease picture and prescribe according to Boger’s method of prescribing. Utilization of modern diagnostic techniques like slit lamp examination in this particular case, helps ascertain the stage of the disease.

CASE:

A 45 year old fisherman reported with a history of injury to the left eye from an iron wire, which occurred while fishing 15 days ago. Thereafter he developed left eye redness, lacrimation, swelling, pain and visual loss. Difficulty in opening the eye especially in light. Pain in the left eye, stitching pain 3+ < light 3+, < cold air2+ and > wamth3+.

The patient had visited an ophthalmologist and received the following treatment (Tab Odoxil, Tab Wysolone, Zoxan Eye Ointment, E/D Mil flox, E/D Predmet for 15 days). He was not responding to the treatment ; he developed Hypopyon (pus in the anterior chamber) which is evident in the photograph.

Note. These are the actual photographs of the patient published with patient’s consent.

O/E : Left Eye (Left eye Photographs – Naked eye appearance and Slit lamp photography) Torch Light Examination: Both lids oedema. Circumcorneal congestion. Photophobia (Homeopathy Treatment for Photophobia). Corneal wound. Hypopion

Traumatic Suppurative Eye - Threat to Vision 1  Traumatic Suppurative Eye - Threat to Vision 2  Traumatic Suppurative Eye - Threat to Vision 3

Slit Lamp Examination: Circumcorneal Congestion. Photophobia. Corneal Laceration at 8 o’clock position. Hypopion (PUS in the anterior chamber) Pupil not reacting to light. (Pupil Fixed) Complicated Cataract.

Traumatic Suppurative Eye - Threat to Vision 4

CLINICAL UNDERSTANDING

Post traumatic suppuration and inflammation like iritis and cataract.

HOMOEOPATHIC UNDERSTANDING

Acute non miasmatic disease with secondary miasmatic expression (Tuberculo-syphilitic)

TREATMENT STRATEGY:

It’s important to understand the disease phenomenon, the stage of disease and define the role of homoeopathy in curing this patient.

STEP 1 Medical Treatment (Acute Remedy → Constitutional Treatment)

Corneal injury with infection → pus in the anterior chamber; iritis → Complicated cataract. The aim is to treat the infection – pus in the anterior chamber should disappear. To reduce the inflammatory signs – (control iritis)

completely quiet the eye – treat infection as well as inflammation.

CAUTION: Progressive hypopion may block the angle of the anterior chamber leading to secondary glaucoma. It is very important to treat the infection rapidly and remove the pus.

STEP 2 Surgical Treatment

Cataract is a sequeale to the inflammation in the eye. It is a hindrance to normal vision. It requires surgical treatment (cataract extraction with iridectomy in order to release the posterior synechae) to restore normal vision. Cataract / Posterior synechae (adhesions) are the sequelae of infection and inflammation which needs surgical treatment. Removal of hard cataract will help to restore the vision at the earliest. Surgical intervention could not be done earlier due to presence of active infection and pus in the eye which can induce Pan Opthalmitis.

Application of The Strategy

Step 1 Medical Treatment

Boger’s Approach (Order of totality construction):

Modalities (Causation, Aggravating and Ameliorating) Sensations and tissue affinity

Totality

1. A/F Injury to Eye

2. Eye Pain < Cold Air

3. Eye Pain > Warmth

4. Pain Stitching.

5. Suppurative Wound

6. Hypopyon (Pus in the anterior chamber)

Repertorisation:

Traumatic Suppurative Eye - Threat to Vision 5

Differential Remedies:

Hepar Sulph, Silicea, Spigelia, Sulphur, Aconite

Hepar Sulph and Silicea are both remedies well known for ailments from injuries. Injuries may undergo a process of suppuration, ie pus formation.

The process is different in both the remedies.

Hepar sulph: Every injury suppurates and there is threatening pus formation.

Quick suppurations. Violence.

Silicea: It has suppurations which are very slow and there would be impending or incomplete abscesses. Only after a dose or two of Silicea will the process be hastened. There may be symptoms of abscess, but it takes long to ripen; it will be indurated.

Cornea is the seat of disease in both the remedies but the difference lies in the pathology – Hepar is known for ulcerations and Silicea for cicatrices / opacification. Both have common modalities like cold aggravation and warm amelioration. The difference lies in the degree of expression. Hepar sulph is known for its violence, rapidity and deeper action. It is highly sensitive in both nature and disease expression. Silicea is known for its torpidity, slowness, delayed, stubborn and incomplete processes at the general expression as well as the suppurative process.

Spigelia:

Affinity for nerves → neuralgia. It produces inflammatory pathologies rather than suppurative pains. Very acute manifestation. Deep seated Inflammatory pains involving cornea, iris and sclera. Neuralgia of eyes, especially where there is great soreness, and one can scarcely bear a touch; affections of the eyeball; eyelids; optic nerve. Pains in eyes, deeply seated in sockets.

Eyes : sore, drawing ache; sunken; feel large; red; flowing tears on affected side; squinting; yellow rings about.

Sulphur:

Generally has psoric dimension → produces more of pruritic and inflammatory eye conditions involving the exterior portions of the eye, ie. Eye Lid, Eye Lashes, Conjuctiva etc. Eye burns; cutting sensation, as from sand; bursting in balls; quivering. Burning sensation, photophobia, opthalmia.

Miasmatic view:

Hepar Sulph generally originates from tubercular miasm and later progresses into syphilitic dimension.

Silicea originates from sycotic miasm and later may advance into tubercular dimension.

Miasmatic study can be done through the expression of disease while it progresses from its origin until the end.

Final Acute Remedy Selction:Hepar Sulph

Criteria of remedy selection relevant to this case:

1. Causation

2. Miasmatic consideration: Tubercular-Syphilitic disease activity, injury which leads to suppuration with rapidity and resulting in post infective state, post inflammatory complication.

3. Pace of development of pathology: Rapid; Injury suppuration despite receiving antibiotics (oral and topical) .

4. Predominant location of disease – corneal and anterior chamber

Remedy (Acute Remedy) Hepar Sulph

(3-5-2010 till 27-6-2011) HEPAR SULPH 200 M 5 PILLS 4 HOURLY

Follow ups :

Pain/Swelling / Lacrymation / Redness / Pus/ Photophobia — 0 –

Hypopion : Disappeared

Traumatic Suppurative Eye - Threat to Vision 6

CONSTITUTIONAL DATA

Mental Dispositional qualities

Calm

Industrious

Hardworking

Physical generals

Chilly patient

Winter cracks

Suppurating injury

Impression: Silicea

According to the ORGANON OF MEDICINE, once the acute inflammatory and infective process is controlled, then case should be followed by deep a acting constitutional remedy.

Note: Although the constitutional remedy itself covered the disease acute totality, it was not prescribed, since the constitutional remedy did not correspond with the depth of pathology and qualitative state of susceptibility.

27-6-2011

SILICEA 200 single dose given and patient was normal, thereafter.

Strategy Step 2 (Surgical Line of Treatment For The Cataract):

Patient’s eye was quiet after homoeopathic treatment which eventually made ground for surgical intervention. This case was under continuous supervision of an ophthalmologist. Seeing the response of homoeopathic medicine, the patient was not put on any pre-operative antibiotic regime by the ophthalmologist. Cataract extraction with PCIOL insertion was done.

Traumatic Suppurative Eye - Threat to Vision 7

Traumatic Suppurative Eye - Threat to Vision 8

 

About the author

Rakesh Gupta

Rakesh Gupta

Dr Rakesh Gupta MD (HOM) BHMS (SMT CMPH Medical College) practices in Mumbai . He had nine years clinical experience in OPD and IPD in a hospital and was a homoeopathic consultant at Satva Homoeopathic Clinic and Masjid Bunder Clinic. He was also junior homoeopathic consultant in the Department of Medicine, Casualty, ICU and Ophthalmology at the M.L.Dhawale Memorial Trust Hospital. From 2004 to the present he has given numerous case presentations. He is currrently Head Of Department Forensic Medicine and Toxicology, Smt C.M.P.H. Medical college, Vile Parle (West), Mumbai & Honorary Homoeopathic Consultant Shree Mumbadevi Homoeopathic Hospital, Vile Parle (West), Mumbai.

12 Comments

Leave a Comment